METHODS OF TREATMENT OF DIABETIC POLYNEUROPATHY

Abstract

Normoglycemia is a key condition of DPN prophylaxis. Research has shown that against the background of 5 years of intensive insulin therapy, DPN is known to have a significant decrease in the frequency of development (by 64%), a decrease in the frequency of detection of nerve fiber conduction disorders (by 44%) and a decrease in the development of autonomic dysfunction (by 53%) [5, 11]. It should be remembered that each patient has a "glycemic ulcer". Pathological processes when the Anashu bulge increases cascade reactions are triggered and lead to the development of DPN [4, 6, 7]. But its duration is considered more important than the degree of hyperglycemia. Prolonged retention of normoglycemia in patients with severe levels of DPN has been found to cause some delay in progressive damage to the peripheral nerves, which is certainly very important, but does not contribute to the recovery of the damaged nerve [13, 14, 15]. Therefore, Additional symptomatic treatment is required to improve the quality of life of patients, especially in the presence of pain syndrome[9].